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Swiss mice fed an AlC1,-mixed (20 mgll00 g body weight) diet for 10 days showed increased lipid peroxidation in liver and kidneys, but not in brain regions and the heart; the possible mechanism of action being through increased generation of reactive oxygen species (ROS). Co-administration of aspinn (1 5 mg/100 g body weight) through diet reduced lipid peroxidation induced by the aluminium salt. It is believed that aspinn reactive intermediates act as scavengers of ROS responsible for the lipid peroxidation.

Palabras clave: aluminium, aspirin, lipid peroxidation, mice.
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Universidad Autónoma del Estado de México
Sistema de Información Científica Redalyc ®
Versión 3.0 | 2017
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